Body odor metabolism map (V20)

Today we release a new curated metabolism map regarding body odor diseases.

The new map alignes around the main inhibited enzymes by body odor diseases.

Beside of that a first receptor linkage has been included.

The map further shows now measured chemical compounds in unnormal ranges of tmau2 and bromhidrosis with elevated TSH.

Obviously in the tmau2 case the measurements align pretty good with the assumption that the intestinal tract metabolism runs out of SAM (S-adenosyl-methionine) by defects in the homocysteine to methionine conversion.

https://drive.google.com/file/d/0B9gtX6As3TJGTDVfNDRfek1WLTA/view?usp=drivesdk

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Bromhidrosis TSH+: Diet protocol + nystatin, nigella sativa + h1 receptor antagonists + medicinal clay improves body odor condition

In our explorative case we were further able to improve the diet protocol as well as finding a new drug/supplement composition. We are taking advantage of following over the counter drugs/supplements: - Nystatin - Nigella sativa (black cumin) capsules - H1 receptor antagonists (loratadin) - medicinal clay The diet protocol was taken from: https://drive.google.com/folderview?id=19hmatFJPil1svc9DRyuRn3S9rUF7yqH_ Dosages (daily) applied were like the following: nystatin: 6x pills (3x at morning, 3x at evening) nigella sativa: >10000mg (5x 400mg capsules before every meal, 5 meals per day) loratadin: 50mg - 100mg daily medicinal clay: 6x 6g (luvos healing earth)

The fecal body odor smell type

As stated in an earlier post, the most prominent reported smell type for TMAU2 patients was the fecal smell type. But not only TMAU positive patients reported such a type of smell, another body odor sufferer type do show the same fecal body odor pattern. Interestingly nearly all of those non TMAU patients report having Irritable Bowel Syndrom (IBS) as well. We took a closer look into the combined bacterial and human metabolism. The central chemical compound involved in the fecal smell seems to be indole. Indole is produced within the bacterial metabolism as a precursor of tryptophan. As major source the bacteria use glycolysis based on sugar, carbohydrates, ... To let the bacteria produce too much indole we found certain criteria: - Tryptophan absorption of the intestinal cells is lower than the serine and glycine absorption - Histidine malabsorption is present - Tyrosine malabsorption is present Why do TMAU2 patients now show a fecal smell, don't they have a choline and

Food diet lists for fecal body odor and Bromhidrosis patients

Based on the data of the USDA Food Composition Database we extrapolated four groups of foods. The respective lists can be found here: https://drive.google.com/folderview?id=0B9gtX6As3TJGa25zX1ZvNmZROUU

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